Glow 70mg: Safety Profile & Research Summary
26 PubMed CitationsExpert ReviewedGMP CertifiedLast Reviewed: April 2026
Preclinical Research Summary (Component-Level)
No peer-reviewed studies have evaluated the GLOW 50/10/10 blend as a co-administered formulation. The studies below summarize key findings for each individual component. For full per-component data tables and clinical references, see the dedicated GHK-Cu, BPC-157, and TB-500 entries.
Component-Level Highlights
| Component | Representative Study | Key Finding |
|---|---|---|
| GHK-Cu | Canapp et al. (2003) — rat ischemic wounds | Wound size ↓ 64.5% vs 28.2% control; ↓ TNF-α, MMP-2/9 |
| GHK-Cu | Pickart & Margolina (2018) — gene-data review | Modulates >4,000 genes; resets expression toward "younger" state |
| BPC-157 | Hsieh et al. (2017) — rat hind-limb ischemia + CAM | 129–152% increased angiogenesis; VEGFR2-Akt-eNOS confirmed |
| BPC-157 | Staresinic et al. (2003) — rat Achilles transection | 10 µg/kg IP: improved AFI scores and load-to-failure 14–72d |
| TB-500 | Philp et al. (2003) — db/db diabetic and aged mice | LKKTETQ fragment promoted dermal repair comparable to full Tβ4 |
| TB-500 (parent Tβ4) | Bock-Marquette et al. (2004) — Nature | Tβ4 activates ILK → cardiac cell migration, survival, repair |
The products offered on this website are furnished for in-vitro studies only. In-vitro studies (Latin: in glass) are performed outside of the body. These products are not medicines or drugs and have not been approved by the FDA to prevent, treat or cure any medical condition, ailment or disease. Bodily introduction of any kind into humans or animals is strictly forbidden by law.
For Laboratory Research Only. Not for human use, medical use, diagnostic use, or veterinary use.
ALL ARTICLES AND PRODUCT INFORMATION PROVIDED ON THIS WEBSITE ARE FOR INFORMATIONAL AND EDUCATIONAL PURPOSES ONLY.
In Vitro / Preclinical Disclaimer: The research findings described above were observed in laboratory (in vitro) and/or animal model studies. These results may not translate to human outcomes. No claims are made regarding human therapeutic applications.
Educational Use Only: This content is provided for informational and educational purposes only. It is not intended as medical advice and should not be used to diagnose, treat, cure, or prevent any disease or medical condition. Always consult qualified healthcare professionals for medical decisions.
References
- Pickart L, Margolina A. Regenerative and Protective Actions of the GHK-Cu Peptide in the Light of the New Gene Data. International Journal of Molecular Sciences. 2018;19(7):1987.
- Sikiric P, et al. Stable Gastric Pentadecapeptide BPC 157, Robert's Stomach Cytoprotection. Current Pharmaceutical Design. 2020;26(25):3024-3044.
- Goldstein AL, Hannappel E, Sosne G, Kleinman HK. Thymosin β4: a multi-functional regenerative peptide. Expert Opinion on Biological Therapy. 2012;12(1):37-51.
- Pickart L, Vasquez-Soltero JM, Margolina A. GHK-Cu may Prevent Oxidative Stress in Skin. Cosmetics. 2015;2(3):236-247.
- Hsieh MJ, et al. Therapeutic potential of pro-angiogenic BPC157 is associated with VEGFR2 activation. Journal of Molecular Medicine. 2017;95(3):323-333.
- Xing Y, Ye Y, Zuo H, Li Y. Progress on the Function and Application of Thymosin β4. Frontiers in Endocrinology. 2021;12:767785.
- U.S. Food and Drug Administration. Certain Bulk Drug Substances for Use in Compounding. FDA.gov. Updated 2023.
- World Anti-Doping Agency. The 2025 Prohibited List. WADA. January 1, 2025.
- Pickart L, Vasquez-Soltero JM, Margolina A. GHK and DNA: Resetting the human genome to health. BioMed Research International. 2014;2014:151479.
- Pickart L, Vasquez-Soltero JM, Margolina A. GHK Peptide as a Natural Modulator of Multiple Cellular Pathways. BioMed Research International. 2015;2015:648108.
- Maquart FX, Pickart L, et al. Stimulation of collagen synthesis by GHK-Cu. FEBS Letters. 1988;238(2):343-346.
- Schlosser N. BPC-157: A Polyproline II Helix Engages SH3 Domains of Src Family Kinases. 2025.
- Chang CH, et al. BPC 157 Enhances the Growth Hormone Receptor Expression in Tendon Fibroblasts. Molecules. 2014;19(12):19066-19077.
- Bock-Marquette I, et al. Thymosin beta4 activates integrin-linked kinase and promotes cardiac cell migration. Nature. 2004;432(7016):466-472.
- Sosne G, Qiu P, Goldstein AL, Wheater M. Biological activities of thymosin beta 4. The FASEB Journal. 2010;24(7):2144-2151.
- Bock-Marquette I, et al. Thymosin beta-4 denotes new directions for anti-aging therapies. International Immunopharmacology. 2023;116:109741.
- Canapp SO Jr, et al. Topical tripeptide-copper complex on healing of ischemic open wounds. Veterinary Surgery. 2003;32(6):515-523.
- Philp D, et al. Thymosin β4 and a synthetic peptide containing its actin-binding domain promote dermal wound repair. Wound Repair and Regeneration. 2003;11(1):19-24.
- Staresinic M, et al. BPC 157 accelerates healing of transected rat Achilles tendon. Journal of Orthopaedic Research. 2003;21(6):976-983.
- Badenhorst T, et al. Effects of GHK-Cu on MMP and TIMP Expression. Journal of Aging Science. 2016;4(3):166.
- Smart N, et al. Thymosin β4 induces adult epicardial progenitor mobilization and neovascularization. Nature. 2007;445(7124):177-182.
- Kuceki G, et al. Enhanced hair regrowth with minoxidil-dutasteride-copper peptides for androgenetic alopecia. JAAD International. 2025;20:38-40.
- Goldstein AL, et al. Thymosin β4: actin-sequestering protein moonlights to repair injured tissues. Trends in Molecular Medicine. 2005;11(9):421-429.
- Sever M, et al. BPC 157 counteracts liver fibrosis. Journal of Physiology and Pharmacology. 2019;70(3):391-400.
- Shah R, et al. Thymosin β4 Prevents Oxidative Stress, Inflammation, and Fibrosis in Liver Injury. Oxidative Medicine and Cellular Longevity. 2018;2018:9630175.
- Sosne G, Dunn SP, Kim C. Thymosin β4 Improves Severe Dry Eye in a Phase 2 Randomized Trial. Cornea. 2015;34(5):491-496.
Related Research Questions
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This content is provided for educational and informational purposes only. Products are furnished for in-vitro studies only and are not medicines, drugs, or supplements. Not approved by the FDA to prevent, treat, or cure any condition.